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gingival overgrowth/glutathione

Povezava se shrani v odložišče
ČlankiKliničnih preskušanjPatenti
5 rezultatov

Altered oxidative status and integrin expression in cyclosporine A-treated oral epithelial cells.

Samo registrirani uporabniki lahko prevajajo članke
Prijava / prijava
OBJECTIVE Cyclosporine A (CsA) is an immunosuppressive agent administered to transplant patients. A well-known reported oral side effect of CsA consumption is gingival overgrowth (GO). Changes in the expression of integrins occurring in the gingiva following CsA treatment have been reported but

Ligneous periodontitis and gingival antioxidant status: report of two cases.

Samo registrirani uporabniki lahko prevajajo članke
Prijava / prijava
Ligneous periodontitis (LP) is a rare periodontal disease in which plasminogen deficiency and fibrin deposition both play a part, resulting in characteristic gingival enlargement and periodontal breakdown. Recent data suggest that oxidant/antioxidant changes are significant in the pathology of oral

Regulation of type I plasminogen activator inhibitor in human gingival fibroblasts with cyclosporine A.

Samo registrirani uporabniki lahko prevajajo članke
Prijava / prijava
OBJECTIVE Cyclosporine A (CsA) is used as an immunosuppressive agent and its prominent side effect is the induction of gingival overgrowth. Type I plasminogen activator inhibitor (PAI-1) has shown to play an important role in CsA-induced gingival overgrowth. However, little is known about whether

Antioxidant activity of blood serum and saliva in patients with periodontal disease treated due to epilepsy.

Samo registrirani uporabniki lahko prevajajo članke
Prijava / prijava
OBJECTIVE The aim of the study was to estimate the activity of chosen antioxidants in blood serum and saliva in patients with periodontal disease treated due to epilepsy. METHODS Twenty-five epileptics and fifteen control persons were involved in the study. The activity of selected endogenous
BACKGROUND Transglutaminase-2 (TGM-2) has been implicated in several fibrotic disorders and can be induced by reactive oxygen species (ROS). Hence, the authors hypothesize that cyclosporin A (CsA) may regulate TGM-2 via ROS, and this regulation may have a role in the pathogenesis of CsA-induced
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