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isopimaric acid/amyloidosis

Povezava se shrani v odložišče
ČlankiKliničnih preskušanjPatenti
2 rezultatov

Suppression of a neocortical potassium channel activity by intracellular amyloid-β and its rescue with Homer1a.

Samo registrirani uporabniki lahko prevajajo članke
Prijava / prijava
It is proposed that intracellular amyloid-β (Aβ), before extracellular plaque formation, triggers cognitive deficits in Alzheimer disease (AD). Here we report how intracellular Aβ affects neuronal properties. This was done by injecting Aβ protein into rat and mouse neocortical pyramidal cells
Transcranial magnetic stimulation (TMS) is fragmentarily reported to be beneficial to Alzheimer's patients. Its underlying mechanism was investigated. TMS was applied at 1, 10 or 15 Hz daily for 4 weeks to young Alzheimer's disease model mice (3xTg), in which intracellular soluble amyloid-β is
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