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lordosis/sesamum indicum
Povezava se shrani v odložišče
14 rezultatov
Activation of the GPR30 receptor promotes lordosis in female mice.
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OBJECTIVE
Estrogens are important effectors of reproduction and are critical for upregulating female reproductive behavior or lordosis in females. In addition to the importance of transcriptional regulation of genes by 17β-estradiol-bound estrogen receptors (ER), extranuclear signal transduction
Absence of a diurnal rhythm in lordosis behaviour induced by oestrogen in gonadectomized rats.
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Gonadectomized rats bearing s.c. Silastic capsules containing crystalline oestradiol-17 beta diluted with cholesterol, or oestradiol-17 beta dissolved in sesame oil were tested for the presence of a diurnal rhythm in the display of lordotic behaviour. In experiment 1, female rats received four
Adolescent cannabinoid treatment negatively affects reproductive behavior in female rats.
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Sex differences in the neurobehavioral effects of chronic cannabinoid exposure suggest that gonadal hormones may modify cannabinoid activity. The current experiment assessed the impact of combined cannabinoid and estradiol treatment in ovariectomized, adolescent female rats on subsequent adult
ERalpha, but not ERbeta, mediates the expression of sexual behavior in the female rat.
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Estrogen has well known effects on sexual behavior, however the role of the estrogen receptors (ER) alpha and beta on sexual behavior remains to be fully determined. This study investigated the individual and co-operative involvement of ERalpha and beta on sexual behaviors in the adult female rat.
Effects of neonatal 17α-ethinyloestradiol exposure on female-paced mating behaviour in the rat.
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Correct perinatal oestrogen levels are critical for sexual differentiation. For example, perinatal exposure to oestrogen causes masculinization and defeminization of the brain in female rats and also induces delayed effects after maturation characterized by early onset of abnormal oestrus cycling.
Progesterone facilitates thrust in female hamsters implanted with estradiol at the medial preoptic area.
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Progesterone (P) facilitates male-like sexual behavior in female hamsters primed with estrogen. Since estrogen plus P activates lordosis in female hamsters and the site of the hormonal action is at the ventromedial hypothalamus (VMH), it is possible that the same hormones act at the same site to
Bisphenol A increases progesterone receptor immunoreactivity in the hypothalamus in a dose-dependent manner and affects sexual behaviour in adult ovariectomized rats.
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Recently, we reported that bisphenol A (BPA), an endocrine disrupter, increased progesterone receptor (PR) mRNA in the preoptic area (POA) in adult ovariectomized rats. In the present study, we examined whether BPA also induced expression of PR proteins in both the POA and the ventromedial
RU486 facilitates or disrupts the sensitization of sexual behaviors by estradiol in the ovariectomized Long-Evans rat: Effect of timecourse.
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An acute injection of estradiol benzoate (EB) to the ovariectomized (OVX) rat activates low levels of lordosis, and subsequent progesterone (P) administration augments lordosis and recruits a complete pattern of sexual behavior including appetitive behaviors (e.g., hops/darts and solicitations).
Inhibition of 5alpha-reductase enzyme or GABA(A) receptors in the VMH and the VTA attenuates progesterone-induced sexual behavior in rats and hamsters.
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Progesterone (P) to the ventromedial hypothalamus (VMH) and the ventral tegmental area (VTA) of ovariectomized (OVX), estradiol benzoate (EB)-primed rats and hamsters produces female sexual behavior similar to that seen in proestrous, receptive rodents. Because P's 5alpha-reduced metabolites can
Behavioral effects of 3 alpha-androstanediol. I: Modulation of sexual receptivity and promotion of GABA-stimulated chloride flux.
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Pregnane neurosteroids may initiate sexual receptivity not only via actions at intracellular receptors, but by affecting gamma-aminobutyric acid (GABA) receptor complexes (GBRs). To investigate whether GBR-mediated actions of an androgenic neurosteroid 5 alpha-androstane-3 alpha, 17 beta-diol (3
Estrogenic action by tris(2,6-dimethylphenyl) phosphate impairs the development of female reproductive functions.
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Developmental exposure to environmental chemicals with estrogen-like activity is suspected to permanently impair women's health. In this study, a mouse model was used to evaluate whether tris(2,6-dimethylphenyl) phosphate (TDMPP), a chemical with a putative estrogen-like action, impairs sexual
Effect of systemic blockade of α(1)-noradrenergic receptors on sex behavior and vaginal-cervical stimulation-induced Fos in female rats.
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It is hypothesized that systemic α₁-noradrenergic antagonists may interfere with the transmission of sensory stimulation, particularly vaginal--cervical stimulation (VCS), which is crucial for reproductive functioning. To determine if α₁-noradrenergic transmission receptor activity is necessary for
Effects of neonatal exposure to the antiprogestin mifepristone, RU 486, on the sexual development of the rat.
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RU 38486 (RU 486, mifepristone) is a potent progesterone receptor antagonist that has been used in humans in the pharmacologic induction of abortion. The effects of exposure to RU 486 during the neonatal period of the rat has not been previously reported. We examined the consequences of such
Neonatal septal lesions prevent behavioral defeminization caused by neonatal treatment with estradiol in female rats.
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Male rats rarely show lordosis, a female sexual behavior, because of strong inhibition of the behavior in the lateral septum. Because neonatal treatment with estradiol (E2) in female rats decreases lordosis, it is believed that the lateral septum is a target of E2 action to defeminize or masculinize
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