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neurogenic inflammation/edema
Povezava se shrani v odložišče
Stran 1 iz 130 rezultatov
Neurogenic inflammation is associated with development of edema and functional deficits following traumatic brain injury in rats.
Samo registrirani uporabniki lahko prevajajo članke
Prijava / prijava
The present study has used capsaicin-induced neuropeptide depletion to examine the role of neurogenic inflammation in the development of edema and functional deficits following traumatic brain injury (TBI). Adult, male rats were treated with capsaicin (neuropeptide-depleted) or equal volume vehicle
Alteration of neuropeptides in the lung tissue correlates brain death-induced neurogenic edema.
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Prijava / prijava
BACKGROUND
Increased intracranial pressure induces neurogenic pulmonary edema (NPE), potentially explaining why only lungs from less than 20% of brain dead organ donors can be used for transplantation. This study investigated the underlying mechanisms of NPE, focusing on neuropeptides, which
Continuous intra-arterial application of substance P induces signs and symptoms of experimental complex regional pain syndrome (CRPS) such as edema, inflammation and mechanical pain but no thermal pain.
Samo registrirani uporabniki lahko prevajajo članke
Prijava / prijava
Substance P is involved in nociception in both the peripheral nervous system and the CNS and has been documented to play a crucial role in the complex regional pain syndrome (CRPS). So far, however, most experimental animal models are restricted to the effect of neurokinin-1 receptor blockers to
Neurogenic inflammation: a first line of defense for the ocular surface.
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Prijava / prijava
Neurogenic inflammation and innate immunity may work together to protect mucosal surfaces, including the ocular surface. When the eye is exposed to pathogens, chemical irritants, or mechanical disruption, neurogenic inflammation is produced through release of neuromodulators, such as substance P and
Facilitated neurogenic inflammation in unaffected limbs of patients with complex regional pain syndrome.
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Prijava / prijava
Pain, edema, increased skin temperature, reddening and trophic changes characterize complex regional pain syndrome (CRPS). Recently, we have been able to show facilitated neurogenic inflammation on the affected limb. In the current study unaffected limbs were examined after resolution of the CRPS
Increased pain and neurogenic inflammation in mice deficient of neutral endopeptidase.
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Prijava / prijava
The complex regional pain syndrome (CRPS) is characterized by enhanced neurogenic inflammation, mediated by neuropeptides. Neutral endopeptidase (NEP) is a key enzyme in neuropeptide catabolism. We used NEP knock out (ko) mice to investigate whether NEP deficiency leads to increased pain behavior
The selective PAC1 receptor agonist maxadilan inhibits neurogenic vasodilation and edema formation in the mouse skin.
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Prijava / prijava
We have earlier shown that PACAP-38 decreases neurogenic inflammation. However, there were no data on its receptorial mechanism and the involvement of its PAC1 and VPAC1/2 receptors (PAC1R, VPAC1/2R) in this inhibitory effect. Neurogenic inflammation in the mouse ear was induced by topical
Prophylactic proopiomelanocortin expression alleviates capsaicin-induced neurogenic inflammation in rat trachea.
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Prijava / prijava
Neurogenic inflammation frequently causes acute plasma leakage in airways and life-threatening pulmonary edema. However, limited strategies are available to alleviate neurogenic inflammation. Proopiomelanocortin (POMC) is the precursor of anti-inflammatory melanocortins, which have been proposed of
Involvement of the tuberomammillary nucleus of the hypothalamus in the modulation of nociception and joint edema in a model of monoarthritis
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Prijava / prijava
Aims: Investigate the involvement of the histaminergic projections from tuberomammillary nucleus (TMN) to the spinal cord in the modulation of nociception and peripheral edema in a model of monoarthritis.
Substance P is associated with the development of brain edema and functional deficits after traumatic brain injury.
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Prijava / prijava
Brain edema and swelling is a critical factor in the high mortality and morbidity associated with traumatic brain injury (TBI). Despite this, the mechanisms associated with its development are poorly understood and interventions have not changed in over 30 years. Although neuropeptides and
Anti-edema action of formoterol in rat trachea does not depend on capsaicin-sensitive sensory nerves.
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Prijava / prijava
The beta 2-adrenergic agonist formoterol has been shown to inhibit plasma extravasation in the respiratory mucosa associated with neurogenic inflammation as well as that caused by histamine or bradykinin. It is unknown whether these effects of formoterol are mediated through an action of sensory
[Relationship between cutaneous temperature and hand edema and allodynia after stroke--the etiology of shoulder-hand syndrome].
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Prijava / prijava
The etiology of shoulder-hand syndrome is as yet unknown. We hypothesized that it may be due to damaged unmyelinated fibers in front of the subscapular muscle. We examined the existence of edema and hypersensitivity to pain in the hands of stroke patients during the subacute stage and their
Inhibition of neurogenic inflammation as a novel treatment for ischemic stroke.
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Prijava / prijava
Each year, 15 million people suffer a stroke, of which 5 million die and 5 million are left permanently disabled. Cerebral swelling is of particular concern following stroke as it accounts for much of the death and disability. However, the mechanisms leading to cerebral swelling are not yet fully
Inhibition of neurogenic inflammation as a novel treatment for ischemic stroke.
Samo registrirani uporabniki lahko prevajajo članke
Prijava / prijava
Each year, 15 million people suffer a stroke, of which 5 million die and 5 million are left permanently disabled. Cerebral swelling is of particular concern following stroke as it accounts for much of the death and disability. However, the mechanisms leading to cerebral swelling are not yet fully
Substance P as a mediator of neurogenic inflammation after balloon compression induced spinal cord injury.
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Prijava / prijava
Although clinical spinal cord injury (SCI) occurs within a closed environment, most experimental models of SCI create an open injury. Such an open environment precludes the measurement of intrathecal pressure (ITP), whose increase after SCI has been linked to the development of greater tissue damage
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