synkinesis/asthenia

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Aberrant reinnervation of facial musculature in a subhuman primate: a correlative analysis of eyelid kinematics, muscle synkinesis, and motoneuron localization.

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Identifikohuni Regjistrohu

A macaque monkey with a preexisting facial nerve injury showed a synkinesis of perioral muscles with blinking and thus provided a serendipitous model for a multiphasic analysis of this common neurologic syndrome. The amplitude of the paretic eyelid in spontaneous and air-puff-induced blinks was

Bilateral oculomotor synkinesis following Miller Fisher syndrome.

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A 64-year-old female presented with ptosis, ophthalmoplegia, ataxia, and weakness. She was diagnosed with Miller Fisher syndrome (MFS) and was treated with plasmapheresis. Six months later, she developed bilateral oculomotor synkinesis, suggesting aberrant regeneration. The pathogenesis of MFS is

Association of Eyelid Position and Facial Nerve Palsy With Unresolved Weakness.

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Identifikohuni Regjistrohu

OBJECTIVE Understanding the prevalence and clinical features of eyelid malpositions in facial nerve palsy (FNP) may inform proper management of patients with FNP and supplement our knowledge of eyelid physiology. OBJECTIVE To describe eyelid malposition in FNP. METHODS In this retrospective cohort

Traumatic intratemporal facial nerve injury: management rationale for preservation of function.

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A retrospective review of 29 cases of intratemporal facial nerve injuries included 18 temporal bone fractures, 7 gunshot wounds, and 4 iatrogenic complications. Surgical exploration confirmed involvement of the fallopian canal in the perigeniculate region in 14 longitudinal and 3 transverse or mixed

Treatment of postparotidectomy salivary fistula with botulinum toxin.

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We report on the successful treatment with botulinum toxin type A local injections of a salivary fistula that occurred after superficial parotidectomy. In a 58-year-old woman, transcutaneous discharge of saliva in the preauricular region had persisted in spite of 2 surgical revisions. Moreover,

Kallmann syndrome patient with gender dysphoria, multiple sclerosis, and thrombophilia.

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One of the challenging issues in patients with complex problems is that the various diseases and their treatment can influence each other and present unusual hurdles in management. We investigated one such complex case. A 34-year-old XY male presented with azoospermia, detected on semen analysis for

Significance of House-Brackmann facial nerve grading global score in the setting of differential facial nerve function.

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OBJECTIVE To determine the clinical significance of the House-Brackmann facial nerve grading scale (HBFNGS) in the setting of differential function along the branches of the facial nerve. METHODS Prospective study of 38 patients with facial palsy who demonstrated differential facial

Hemifacial spasm: treatment by microvascular decompression of the facial nerve at the pons.

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Identifikohuni Regjistrohu

Hemifacial spasm is a syndrome of intermittent and tonic unilateral facial muscular contraction; mild facial weakness on the same side is also frequently present. Hemifacial spasm can be differentiated clinically from habit spasms, blepharospasm, facial synkinesis following Bell's palsy, facial

Atypical parkinsonism in a family of Portuguese ancestry: absence of CAG repeat expansion in the MJD1 gene.

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Identifikohuni Regjistrohu

We studied the clinical features and molecular genetics of a family, afflicted with a form of atypical parkinsonism, originating from the Madeira Islands of Portugal. We examined four affected individuals and reviewed clinical information on one other affected family member. Mean age at onset was 31

Hemifacial spasm: clinical and electrophysiologic observations.

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Identifikohuni Regjistrohu

Twenty-three patients with hemifacial spasm were studied clinically and electrodiagnostically. Seven patients had mild facial weakness. All patients had clinical evidence of synkinesis, which often varied considerably. Facial nerve conduction and blink reflex latencies were normal. Facial synkinesis

Rehabilitation strategies for facial nerve injuries.

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Many treatment techniques, including exercise, electrical stimulation, biofeedback, and neuromuscular retraining, have been described for the treatment of patients with facial paresis. The degree of nerve injury determines the recovery of the facial muscles. Patients with a Sunderland third-degree

[Surgical treatment of hemifacial spasm associated with tortuous vertebrobasilar system (author's transl)].

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Identifikohuni Regjistrohu

Surgical treatment of hemifacial spasm associated with tortuous vertebrobasilar system was reported. A patient was 63-year-old female, who first experienced mild and intermittent muscle twitching around her left eye twenty years prior to admission. Five years later, the twitching extended to all the

What happens in the other eye? Blink reflex alterations in contralateral side after facial palsy.

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Identifikohuni Regjistrohu

Alterations in blink reflex excitability may occur in the contralateral side (CLS) and in the symptomatic side after peripheral facial palsy (PFP). In this study, the alterations of blink reflex in CLS were evaluated in cases with PFP who showed "three different types" of recovery. For this purpose,

Good correlation between original and modified House Brackmann facial grading systems.

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Identifikohuni Regjistrohu

OBJECTIVE Subjective scales of facial function were plagued with reporting variations until the House-Brackmann scale was described in 1985. Despite its utility, weaknesses were identified, including noninclusion of synkinesis phenomena and insensitivity to segmental weakness. Therefore, the scale

Treatment of Bell palsy with prednisone: a prospective, randomized study.

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Identifikohuni Regjistrohu

Two hundred thirty-nine patients with Bell palsy were randomly distributed into prednisone-treated and control groups. Patients were followed until complete recovery or for 1 year. In the steroid-treated and control groups, respectively, incomplete recovery of facial strength occurred in 12 percent

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