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glomerulonephritis/kalium
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Sida 1 från 102 resultat
Voltage-gated potassium channel Kv1.3 blocker as a potential treatment for rat anti-glomerular basement membrane glomerulonephritis.
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The voltage-gated potassium channel Kv1.3 has been recently identified as a molecular target that allows the selective pharmacological suppression of effector memory T cells (T(EM)) without affecting the function of naïve T cells (T(N)) and central memory T cells (T(CM)). We found that Kv1.3 was
Contribution of the distal tubule to potassium excretion in experimental glomerulonephritis.
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The renal adaptations that maintain potassium homeostasis in diffuse forms of glomerular disease are not well defined. Thus, handling of potassium by superficial nephron segments was examined in a rat model of antiglomerular basement membrane nephritis. Sampling the same nephron successively from
Studies of potassium secretion in glomerulonephritis.
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[Change of potassium and sodium homeostasis in different stages of renal insufficiency in patients with glomerulonephritis].
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[Relation between blood potassium & the electrocardiogram in glomerulonephritis].
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[Renal excretion of sodium, potassium and chlorides in chronic glomerulonephritis with impaired renal function].
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[Effect of lasix on excretion of aldosterone, sodium, potassium and diuresis in children with chronic glomerulonephritis and pyelonephritis].
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[Comparison between electrocardiographic changes and the level of blood potassium in different stages and clinical forms of the course of diffuse glomerulonephritis].
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[The participation of the kallikrein-kinin system in edema formation in glomerulonephritis].
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Involvement of the kallikrein-kinin system in the pathogenesis of renal edemas may be mediated by increase of vascular permeability, proteinuria, diuresis and natriuresis. Proceeding from these points, in 27 patients with morphologically proved chronic glomerulonephritis and the nephrotic syndrome,
Effects of indomethacin on the renal function and renin-aldosterone system in chronic glomerulonephritis.
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The effects of indomethacin on plasma renin activity (PRA), plasma and urine aldosterone levels and on renal function were studied in 37 patients with chronic glomerulonephritis (GN). Indomethacin produced a significant decrease in PRA, in plasma and urinary aldosterone levels and an increase in
[Immune complex glomerulonephritis associated with pulmonary tuberculosis].
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A 32 year old man was admitted for dyspnea, hemoptysis, macroscopic hematuria, hypertension (140/100), peripheral edema and hemodynamic decompensation. Lung Xrays revealed pulmonary edema and a cavity in the left apex. Laboratory determinations revealed an altered renal function with increased
[The effects of strict dietary salt restriction on blood pressure and proteinuria in chronic glomerulonephritis patients.].
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OBJECTIVE
To evaluate the effects of strict dietary salt restriction on blood pressure and proteinuria in chronic glomerulonephritis (CGN) patients.
METHODS
From October 2007 to April 2009, 32 CGN inpatients were enrolled. Among them, 15 patients followed a strict dietary salt restriction menu
Urinary excretion of prostaglandins (PGE2 and PGF2 alpha) and kallikrein in acute glomerulonephritis.
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We studied prostaglandins and kallikrein urinary excretion in 14 children with acute poststreptococcal glomerulonephritis within 48 hours of hospital admission (period A), and again, 4-6 weeks later, when they were clinically recovered (period B). Seventeen apparently healthy children were studied
Efficacy of low-dose spironolactone on top of angiotensin receptor blockade in patients with glomerulonephritis.
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UNASSIGNED
Previous studies have shown that aldosterone antagonists have a proteinuria-lowering effect in patients with proteinuria and progressive proteinuric disease not adequately controlled by the use of angiotensin receptor blockers (ARBs). Aldosterone antagonists, in combination with ARBs,
[Effects of glucose ingestion on urinary excretion of electrolytes: comparison between patients with chronic glomerulonephritis and diabetes mellitus].
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We investigated the effects of ingesting 75g of glucose on urinary excretion of electrolytes in patients with chronic glomerulonephritis (CGN) and diabetes mellitus (DM) over a 4-hour period. Creatinine clearance did not change in patients with either disease following glucose ingestion. Fractional
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