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laryngismus/hypoxia
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Laryngospasm and hypoxia after intramuscular administration of ketamine to a patient in excited delirium.
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An advanced life support emergency medical services (EMS) unit was dispatched with law enforcement to a report of a male patient with a possible overdose and psychiatric emergency. Police restrained the patient and cleared EMS into the scene. The patient was identified as having excited delirium,
Prevention and Treatment of Laryngospasm in the Pediatric Patient: A Literature Review.
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Laryngospasm is an exaggeration of a protective reflex that prevents aspiration of foreign objects into the lower airway (eg, during swallowing). This results in complete or partial closure of the glottis, and impedance or total obstruction of airflow to the trachea and lungs. Often, the resulting
Severe laryngospasm without intravenous access--a case report and literature review of the non-intravenous routes of administration of suxamethonium.
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Severe laryngospasm may occur during inhalational induction of paediatric patients. Effective and rapid treatment of this complication is extremely important to prevent severe hypoxia. The treatment of choice is intravenous suxamethonium if muscle relaxation is desired. However, in the absence of
Modification of laryngospasm in response to changes in PaCO2 and PaO2 in the cat.
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In order to study the effects of PaCO2 and PaO2 on the laryngeal closure reflex, changes in laryngeal resistance of the isolated cat larynx were measured before and during the stimulation of the superior laryngeal nerve (SLN) at various levels of PaCO2 and PaO2. The results showed that laryngeal
Postoperative negative pressure pulmonary edema following repetitive laryngospasm even after reversal of neuromuscular blockade by sugammadex: a case report.
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Laryngospasm, an occlusion of the glottis, can occur at any time during anesthesia, and is associated with serious perioperative complications such as hypoxia, hypercabia, aspiration, bronchospasm, arrhythmia, prolonged recovery, cardiac collapse, and eventually catastrophic death. Importantly,
[Laryngospasm].
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OBJECTIVE
Airways management is fundamental for anesthesiologists, especially during induction of anesthesia and after extubation, when laryngeal spasm is more common. The anesthesiologist should know pharyngeal-laryngeal physiology and the risk factors for airways obstruction, since this is a
Laryngospasm-induced pulmonary edema: case report.
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Laryngospasm during the emergent phase of anesthesia is a respected complication well known to any PACU nurse. One complication of laryngospasm is noncardiac pulmonary edema (NCPE). NCPE can be a catastrophic complication of anesthesia. A case report is presented to illustrate the signs and symptoms
Pulmonary edema following postoperative laryngospasm: case reports and review of the literature.
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Pulmonary edema that follows upper airway obstruction may occur in a variety of clinical situations. The predominant mechanism is forced inspiration against a closed or occluded glottis, inducing large intrapleural and transpulmonary pressure gradients favoring the transudation of fluid from the
Ictal laryngospasm monitored by video-EEG and polygraphy: a potential SUDEP mechanism.
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A 56-year-old man with refractory bitemporal epilepsy was monitored in the Epilepsy Monitoring Unit (EMU). In a video-EEG captured seizure, brief oroalimentary automatisms were followed by increased inspiratory effort, accompanied by prominent, visible tracheal movements and audible inspiratory
Different laryngeal responses during respiratory arrest produced by hypoxia withdrawal, thiopentone, ketamine, and lidocaine in cats.
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The changes in laryngeal resistance (LR) during respiratory arrest produced by withdrawal of hypoxia stimulation and administration of various respiratory depressants were studied in 14 spontaneously breathing, anesthetized cats (11 cats with alpha-chloralose and three cats with halothane).
Intubation following high-dose rocuronium in a cat with protracted laryngospasm.
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METHODS
An 11-year-old spayed female domestic shorthair cat with a history of laryngospasm at induction of general anesthesia presented for dental evaluation and treatment. The cat was premedicated with hydromorphone (0.05 mg/kg) and alfaxalone (0.5 mg/kg) intravenously, pre-oxygenated for 5 mins (3
Laryngospasm from the anesthesiologist's viewpoint.
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Laryngeal spasm is a problem constantly confronting the anesthetist. It can be serious and may produce fatal cerebral or cardiac complications. Etiologic agents include primary vagal hypertonicity, anoxemia, and painful stimulation of whatever source. Laryngeal spasm must be differentiated from
Fentanyl-induced chest wall rigidity and laryngospasm in preterm and term infants.
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OBJECTIVE
To assess the occurrence of muscle rigidity after fentanyl administration in premature and term neonates.
METHODS
Prospective case series, observational study.
METHODS
A university hospital neonatal intensive care unit.
METHODS
8/89 preterm and term infants (25-40 wks gestational age) who
Evaluation of near-infrared spectroscopy under apnea-dependent hypoxia in humans.
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In this study we investigated the responsiveness of near-infrared spectroscopy (NIRS) recordings measuring regional cerebral tissue oxygenation (rSO2) during hypoxia in apneic divers. The goal was to mimic dynamic hypoxia as present during cardiopulmonary resuscitation, laryngospasm, airway
The risk factors for hypoxemia in children younger than 5 years old undergoing rigid bronchoscopy for foreign body removal.
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BACKGROUND
Removal of an airway foreign body (FB) is usually performed by rigid bronchoscopy under general anesthesia, but the choice of anesthesia and ventilation techniques varies greatly among anesthesiologists and institutions. Hypoxemia is the most commonly observed adverse event during rigid
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