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pro renin/stroke

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Soluble (Pro)renin Receptor in Elderly Chronic Heart Failure Patients

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Overactivation of renin-angiotensin system (RAS) is one of the main pathophysiological features in the evolution of chronic heart failure (CHF). The (pro)renin receptor ((P)RR) represents an important player in a tissue renin-angiotensin system (tissue RAS), which mediates tissue injury through

(Pro)renin Receptor Blockade Ameliorates Cardiac Injury and Remodeling and Improves Function After Myocardial Infarction.

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BACKGROUND The (pro)renin receptor [(P)RR] is implicated in the pathogenesis of cardiovascular disease. We investigated the effects of (P)RR blockade after myocardial infarction (MI) in a mouse coronary-ligation model. RESULTS Mice underwent sham control surgeries (n = 8) or induction of MI followed

Critical roles of (pro)renin receptor-bound prorenin in diabetes and hypertension: sallies into therapeutic approach.

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High plasma prorenin levels in diabetic patients predict microvascular complications, but the mechanism of the connection between them has remained unclear. (Pro)renin receptors were recently found in the human kidney, and their distribution in various organs, including the heart, has been
Stroke is one of the major medical burdens in industrialized countries. Animal experiments indicate that blockade of the angiotensin AT1 receptor (AT1R) improves neurological outcome after cerebral ischemia. These protective effects are partially mediated by the angiotensin AT2 receptor (AT2R). The

ACE2/ANG-(1-7)/Mas pathway in the brain: the axis of good.

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The last decade has seen the discovery of several new components of the renin-angiotensin system (RAS). Among them, angiotensin converting enzyme-2 (ACE2) and the Mas receptor have forced a reevaluation of the original cascade and led to the emergence of a new arm of the RAS: the ACE2/ANG-(1-7)/Mas
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