anetoderma/amyloid

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NakalaMajaribio ya klinikiHati miliki

Chagua aina ya aina

5 matokeo

[Localized nodular amyloidosis cutis with anetoderma].

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Ingia / Ingia

Fibrillin immunoreactive fibers constitute a unique network in the human dermis: immunohistochemical comparison of the distributions of fibrillin, vitronectin, amyloid P component, and orcein stainable structures in normal skin and elastosis.

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Ingia / Ingia

Fibrillin, a 350-kD glycoprotein, was recently localized to elastin-associated 10 nm microfibrils. Here, the distribution of fibrillin immunoreactivity was determined in normal skin in individuals of different ages and in lesions of solar elastosis or anetoderma. It was compared with the

[Pseudoscleroderma and sclerodermiform states].

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Ingia / Ingia

Pseudo-scleroderma should not be confused with true scleroderma, the prognosis of which is unpredictable and often serious. Progressive acrosclerosis must be differentiated from Raynaud's disease, congenital or hereditary disorders of unknown aetiology: Werner's syndrome, acrogeria and progeria;

The invisible dermatoses.

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Ingia / Ingia

It is understandable that clinically normal skin may show abnormalities when examined with the light microscope, but paradoxical that biopsy of a clinically significant skin disorder may show a histologic picture that looks like normal skin. From the perspective of the dermatopathologist, the

Skin pathology findings in a cohort of 1500 adult and elderly subjects.

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Ingia / Ingia

BACKGROUND No extensive studies are available in the literature on the eventual skin pathology induced by neurologic or systemic diseases in elderly individuals. Other factors, such as health and hygiene, socioeconomic status, and climate can also play an important role. METHODS Fifteen-hundred

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Herbal & Natural Medicine

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