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butea braamiana/triglyceride

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NakalaMajaribio ya klinikiHati miliki
6 matokeo

Chronic toxicity study of Butea monosperma (Linn.) Kuntze seeds in albino rats.

Watumiaji waliosajiliwa tu ndio wanaweza kutafsiri nakala
Ingia / Ingia
In the present study, toxic effects of powder of seeds of Butea monosperma (Linn.) Kuntze were evaluated for a period of 3 months in albino rats. Control group received distilled water. The powder suspension was orally given to the treated group at a dose of 800 mg/kg/day for 90 days. Parameters
We measured a vast range of parameters, in an attempt to further elucidate previously claimed antihyperglycemic activity of Butea monosperma. Our study clearly negates the possibility of antidiabetic activity by inhibited gastrointestinal enzyme action or by reduced glucose absorption. Reduction of
Daily treatment of alloxan-induced diabetic animals with 50% ethanolic extract of B. monosperma flowers (BMEE) for 45 days significantly lowered blood glucose level thereby preventing steep onset of hyperglycemia which was observed after alloxan administration and maintained body weight and blood
Butea monosperma (Lam.) (family: Fabaceae) popularly known as 'Palas' or 'fire of forest' has been used traditionally as a hepatoprotective agent. This study evaluated the hepatoprotective and antitumorigenic properties of the aqueous extract and butanol fractions of B. monosperma flowers in animal

Antihyperglycemic and antioxidant effect of hydroethanolic extract of Butea monosperma bark in diabetic mice.

Watumiaji waliosajiliwa tu ndio wanaweza kutafsiri nakala
Ingia / Ingia
The antihyperglycemic, antihyperlipidemic and antioxidative properties of hydroethanolic extract of Butea monosperma bark were investigated in alloxan-induced diabetic mice. Alloxan administration resulted in higher blood glucose level and reduced hepatic glycogen content as compared to normal
The possible protective effect of ethanolic extract of B. monosperma leaves (BMEE) on diabetes and diabetes-induced oxidative stress was evaluated in alloxan (ALXN)-induced diabetic male adult mice. Experimental animals were divided into three groups viz., I, II, and III. Diabetes mellitus (DM) was
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