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clausenamide/cerebral infarction

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NakalaMajaribio ya klinikiHati miliki
3 matokeo
OBJECTIVE To observe the effect and mechanism of clausenamide on the expression of Bcl-2 and apoptosis after focal cerebral ischemia/reperfusion in renovascular hypertensive rats. METHODS Seventy-five renovascular hypertensive rats were randomly divided into three groups (25 in each group):

The effects of (-)clausenamide on functional recovery in transient focal cerebral ischemia.

Watumiaji waliosajiliwa tu ndio wanaweza kutafsiri nakala
Ingia / Ingia
The effects of (-)clausenamide (clau) on spatial cognitive functions and hippocampal long-term potentiation (LTP) after transient focal cerebral ischemia in rats were investigated. Four weeks after middle cerebral artery occlusion, Morris water maze tasks demonstrated that 2 h of transient forebrain

(-)-Clausenamide alleviated ER stress and apoptosis induced by OGD/R in primary neuron cultures

Watumiaji waliosajiliwa tu ndio wanaweza kutafsiri nakala
Ingia / Ingia
Objective: The endoplasmic reticulum stress (ERS) and ERS-related neuronal apoptosis contribute to the cerebral ischemia/reperfusion (I/R) injury. (-)-Clausenamide has been reported to be nootropic and improve learning and memory in
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