esophageal achalasia/glutathione

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NakalaMajaribio ya klinikiHati miliki

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4 matokeo

Transport of glutathione into the nucleus.

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Ingia / Ingia

The tripeptide thiol glutathione (GSH) is present in the nucleus of plant and animal cells. However, the functions of GSH in the nucleus remain poorly characterised. GSH appears to become sequestered in the nucleus at the early stages of the cell cycle. As part of our search for proteins that may be

Intracellular ROS level is increased in fibroblasts of triple A syndrome patients.

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Ingia / Ingia

Triple A syndrome is named after the main symptoms of alacrima, achalasia, and adrenal insufficiency but also presents with a variety of neurological impairments. To investigate the causes of progressive neurodegeneration, we examined the oxidative status of fibroblast cultures derived from triple A

Deficiency of ALADIN impairs redox homeostasis in human adrenal cells and inhibits steroidogenesis.

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Ingia / Ingia

Triple A syndrome is a rare, autosomal recessive cause of adrenal failure. Additional features include alacrima, achalasia of the esophageal cardia, and progressive neurodegenerative disease. The AAAS gene product is the nuclear pore complex protein alacrima-achalasia-adrenal insufficiency

Compensation for chronic oxidative stress in ALADIN null mice.

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Ingia / Ingia

Mutations in the AAAS gene coding for the nuclear pore complex protein ALADIN lead to the autosomal recessive disorder triple A syndrome. Triple A patients present with a characteristic phenotype including alacrima, achalasia and adrenal insufficiency. Patient fibroblasts show increased levels of

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Herbal & Natural Medicine

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