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demecolcine/enflamasyon

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NesneKlinik denemelerPatentler
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[Effect of urethane, demecolcine and podophyllotoxin on experimental inflammation].

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[The influence of ethyl urethane, demecolcine and podophyllotoxin on experimental inflammation in rats].

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Agents with periodontal regenerative potential regulate cell-mediated collagen lattice contraction in vitro.

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A variety of pharmaceutical agents has been proposed for use in periodontal therapy to inhibit loss of alveolar bone and to promote regeneration of tissues lost to disease. The purpose of this study was to determine the effects of such agents on periodontal cell-mediated gel contraction, an in vitro
Migration inhibitory factor-related protein 8 (MRP8) and MRP14, two S-100-like Ca(2+)-binding proteins, have been described in cells of the epithelial lineage where they are either expressed constitutively (e.g. by mucosal squamous epithelium) or induced during disease (e.g. in keratinocytes during
Natural compounds played an important role for prevention and treatment of the disease as well as are the important compounds for the design of the new bioactive compounds. In this study, eight tropolone alkaloids were isolated from Colchicum kurdicum including colchicoside, 2-demethyl colchicine,

Adhesion of U-937 monocytes induces cytotoxic damage and subsequent proliferation of cultured human mesangial cells.

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In an in vitro model of monocyte adhesion to glomerular cells, U-937 myelomonocytic leukemia cells irreversibly bind to human mesangial cell monolayers. Adhesion is enhanced in mesangial cells proliferating in response to fetal bovine serum, and in the presence of several cytokines and vasoactive

Inhibition of rat perirenal preadipocyte differentiation.

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The process of adipose differentiation uniquely endows fat cells to accrue triacylglycerols under conditions of nutrient energy surfeit and to release fatty acids during energy deprivation. The object of this investigation was to study influences on this process in perirenal preadipocytes, grown in

Proinflammatory cytokines increase glial fibrillary acidic protein expression in enteric glia.

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BACKGROUND Enteric glia protect the integrity of the gut, as loss of enteric glial fibrillary acidic protein (GFAP) positive (+) glia leads to a haemorrhagic jejunoileitis. Crohn's disease (CD) and necrotising enterocolitis (NEC) show pathological changes in enteric glia. Therefore, factors
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