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Anti-inflammatory therapy in an ovine model of fetal hypoxia induced by single umbilical artery ligation.

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Perinatal morbidity and mortality are significantly higher in pregnancies complicated by chronic hypoxia and intrauterine growth restriction (IUGR). Clinically, placental insufficiency and IUGR are strongly associated with a fetoplacental inflammatory response. To explore this further, hypoxia was

Maternal sildenafil impairs the cardiovascular adaptations to chronic hypoxemia in fetal sheep

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Key points: Fetal growth restriction induces a hemodynamic response which aims to maintain blood flow to vital organs such as the brain, in the face of chronic hypoxemia Maternal sildenafil treatment impairs the hypoxemia-driven

Chronic fetal hypoxia increases activin A concentrations in the late-pregnant sheep.

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OBJECTIVE To determine whether activin A concentrations are altered in chronic fetal hypoxemia and intrauterine fetal growth restriction (IUGR). METHODS In vivo animal experimental model. METHODS Department of Physiology, Monash University. METHODS Chronically catherised fetal sheep in late

Vascular anomalies of the umbilical cord. II. Perinatal and pediatric implications.

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Among 62 cases with single umbilical artery and 305 with velamentous insertion of the umbilical cord, we found a high incidence of congenital malformations in both groups, highest in the SUA-group. No organ system seemed to be more affected than the others. In both groups there was a high incidence

The effects of low-dose endotoxin on the umbilicoplacental circulation in preterm sheep.

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OBJECTIVE In the present study we examined the effects of low-dose endotoxin (lipopolysaccharides, LPS) on continuously recorded umbilical blood flow. METHODS Twenty fetal sheep were catheterized at a gestational age of 107 +/- 1 days. A flow probe was placed around either the common umbilical

Angiogenesis in villous chorangiosis observed by ultrastructural studies.

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Chorangiosis is microscopically designated as more than ten terminal capillaries within the villous stroma of the placenta and is mostly related to chronic fetal hypoxia. However, the histogenetic relationship between increased number of terminal villous capillaries and chronic hypoxia has not yet

Arteriovenous ECMO for neonatal respiratory support. A study in perigestational lambs.

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A study was undertaken to investigate the applicability of the arteriovenous mode of perfusion for partial support of neonatal respiration. Perigestational lambs, delivered by cesarean section, served as the animal model of respiratory distress. Arteriovenous flow was accomplished between a single

[Pathological analysis of 76 placentae in fetal distress].

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The result of pathological analysis of 76 placentae and their appendices from clinically diagnosed fetal distress in year 1985-1989 is here presented. The findings could be classified into 4 groups; lesions due to disturbance of maternal blood flow and villous ischemia or hypoxia; retardation of

Effects of antenatal melatonin therapy on lung structure in growth-restricted newborn lambs.

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Oxidative stress arising from suboptimal placental function contributes to a multitude of pathologies in infants compromised by fetal growth restriction (FGR). FGR infants are at high risk for respiratory dysfunction after birth and poor long-term lung function. Our objective was to investigate the

Effects of Antenatal Melatonin Treatment on the Cerebral Vasculature in an Ovine Model of Fetal Growth Restriction.

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Chronic moderate hypoxia, such as occurs in fetal growth restriction (FGR) during gestation, compromises the blood-brain barrier (BBB) and results in structural abnormalities of the cerebral vasculature. We have previously determined the neuroprotective and antioxidant effects of maternal

Fetal Growth Restriction Alters Cerebellar Development in Fetal and Neonatal Sheep.

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Fetal growth restriction (FGR) complicates 5-10% of pregnancies and is associated with increased risks of perinatal morbidity and mortality. The development of cerebellar neuropathology in utero, in response to chronic fetal hypoxia, and over the period of high risk for preterm birth, has not

Morbidity, mortality, and placental pathology in excessively long umbilical cords: retrospective study.

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The purpose of this study was to compare specific fetal, maternal, and placental factors, including neonatal morbidity and mortality, in infants with umbilical cords (UCs) of normal length to the same factors in infants with excessively long umbilical cords (ELUCs). We performed an 18-year

Umbilical artery thrombosis: Two case reports.

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The umbilical cord is the way to exchange gas, supply nutrients, excrete metabolized. Thrombosis of the umbilical cord leads to fetal hypoxia, which jeopardizes fetal health and can cause fetal death. Umbilical vessel thrombosis, which is rarely reported, is difficult to detect

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