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asbestosis/hypoxia

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8 结果

[Hypoxemia and acid-base balance in patients with asbestosis].

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We report a case of severe pulmonary fibrosis in a patient exposed to asbestos and other natural and man-made mineral fibers (MMMF) over a period of time. A 63-year-old man was admitted to our hospital because of progressive dyspnea and severe hypoxemia with hypercapnea. Mineral fibers recovered by
Examination of 72 individuals (including 62 workers of asbestos production and 10 silicosis patients) revealed that asbestos dust primarily causes hypoxemia followed by pulmonary ventilation disorders and characteristic X-ray signs of asbestosis. Hypoxemia is associated by reliable changes in
Environmental asbestos exposure is related to diffuse pleural disease (thickening and calcification) and restrictive pulmonary disease. To assess cardiac autonomic system, we investigated the time domain heart rate variability (HRV) by Holter monitoring and their correlation with pulmonary function
Early changes under exposure to chrysotile-asbestos dust include light arterial hypoxemia, lower bronchial permeability of small bronchi. Early asbestosis and dust bronchitis manifest with impaired pulmonary ventilation of mixed type, that is associated with moderate arterial hypoxemia in dust

Lung disease at high altitude.

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The lungs are a delicate interface between the atmosphere and our bodies across which oxygen diffuses from the air we breathe to the blood which carries oxygen to the cells and mitochondria. In healthy lungs at sea level where there is a surfeit of oxygen, this process occurs easily, whereas, in

[Treatment possibilities and treatment results in pneumoconioses].

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Some types of pneumoconiosis, such as asbestosis, are characterized by marked restrictive functional patterns. Treatment is begun when definite arterial hypoxemia appears, since the inhalation of oxygen clearly lowers pulmonary artery pressure. It is also important that the onset of concomitant

Amiodarone pneumonitis diagnosed by gallium-67 scintigraphy.

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A 75-year-old man presented with fever, dyspnoea, chest pain and findings suggestive of severe pneumonia including hypoxia and bilateral chest infiltrates. He was given antibiotics without significant effect. He took long-term amiodarone therapy for persistent atrial arrhythmias and also had a
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