atrioventricular block/seizures

Differentiation between cardiac and neurological origin of syncope may be challenging. Prolonged cerebral hypoxia secondary to cardiac arrhythmias may lead to epileptic seizures. Moreover, partial epileptic seizures by themselves can trigger cardiac arrhythmias. Herein, we present a case of partial

A 43-year-old man was admitted to our hospital presenting with seizures and syncope. He had a history of a cold with a fever of 39°C occurring three days earlier. Electrocardiography (ECG) showed complete atrioventricular block (AV block) with a maximum pause of 32 seconds, for which temporary

BACKGROUND Synthetic cannabinoid intoxication has become difficult to diagnose and manage in the United States, in part due to varying clinical effects within this heterogeneous group of compounds. METHODS A 38-year-old man was admitted with altered mental status and bradycardia. He demonstrated

The identification of patients with a diagnosis of seizure disorder who are also at risk for clinically significant bradycardia and/or tachycardia may require long-term cardiac rhythm monitoring. Noninvasive, continuous, outpatient cardiac rhythm monitoring may be useful for such clinical scenarios.

Trifascicular block, which consists of impaired conduction in the three main fascicles of the ventricular conduction system, may progress to high-grade or complete atrioventricular block. It rarely occurs in children with a structurally normal heart. We report a case of trifascicular block in a

The frequency and type of seizure-induced cardiac arrhythmias is presented in light of 36 partial-complex seizures recorded with simultaneous EEG/ECG. An average increase in heart rate of 35% (sinus tachycardia) was observed in 60% of patients (22/36). Bradycardia was demonstrated in 17% (6/36) due

Syncope is a condition often misdiagnosed as epilepsy. Syncope caused by cardiac disturbance is a life-threatening condition and accurate diagnosis is crucial for patient outcome. We present a case study of a 71-year-old woman who was referred to our epilepsy centre with a diagnosis of refractory

A 64-year-old man ingested 4500 mg of maprotiline hydrochloride. He developed major motor myoclonic seizures, first-degree AV block, intraventricular conduction delay, hypotension, and urinary retention. Myoclonic seizures have not been previously reported with maprotiline toxicity.