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UNASSIGNED
When a patient with neovascular age-related macular degeneration or diabetic macular edema does not respond to an initial anti-vascular endothelial growth factor agent, usually after several injections, ophthalmologists may switch to another anti-vascular endothelial growth factor agent.
Cerebral edema (CE) is the commonest cause of morbidity and mortality during the first day of treatment for (DKA) in pediatric patients, but if neurological deterioration occurs during treatment of diabetic ketoacidosis, other possibilities must also be considered. We present a 13 month old girl
We report a 48-year-old woman who developed convulsive seizures and cerebral atrophy after recovery from fulminant hepatitis B with coma and cerebral edema at the acute stage. Neurological disturbances and cerebral signs are rare sequelae of fulminant hepatic failure (FHF); only a few cases have
The purpose of this study was to investigate the characteristics of microcystic macular edema (MME) determined from the en face images obtained by an adaptive optics (AO) fundus camera in patients with autosomal dominant optic atrophy (ADOA) and to try to determine the mechanisms underlying the
The senile patients with laryngeal diseases were subjected to phonatory and respiratory function studies. Each patient was examined laryngologically and phoniatrically. The patients were divided into two groups, according to the changes observed in the larynx: as edema and atrophy of the vocal
OBJECTIVE
To report a case of spontaneous bleb formation in a presumed pellucid marginal corneal degeneration with acute hydrops.
METHODS
A 51-year-old man reported sudden ocular pain with a lower conjunctival mass in his left eye. A clinical examination was performed to investigate this
A 26-year-old woman with gyrate atrophy of the choroid and retina and hyperornithinemia lost central visual acuity because of cystoid macular edema associated with epiretinal membrane formation. Dietary manipulation over a 5-month interval reduced the plasma ornithine levels 37% but did not halt the
Purpose: Gyrate atrophy (GA) of the fundus is a rare, progressive metabolic disease secondary to the deficiency of the pyridoxal phosphate-dependent enzyme, ornithine aminotransferase. GA may lead to cystoid macular edema (CME) resulting from chronic inflammation. We aimed to report a child
OBJECTIVE
To analyze the importance f optical coherence tomography (OCT) to diagnose the cystoid macular edema in a case of gyrate atrophy.
METHODS
Observational case report.
METHODS
A 12-year-old boy presenting with gyrate atrophy of the choroid and retina underwent ophthalmologic, clinical, and
BACKGROUND
Recently, we characterized a Gamma Knife® radiation necrosis mouse model with various magnetic resonance imaging (MRI) protocols to identify biomarkers useful in differentiation from tumors. Though the irradiation was focal to one hemisphere, a contralateral injury was observed that
These results show that infusion edema produces a significant increase in brainstem tissue water similar to the distribution seen after traumatic injury. The increased brainstem water of 0.24 cc is equivalent to a brainstem volume rise of 7.26% and was sufficient to cause a marked reduction in the
Non-lethal macular diseases greatly impact patients' life quality, and will cause vision loss at the late stages. Visual inspection of the optical coherence tomography (OCT) images by the experienced clinicians is the main diagnosis technique. We proposed a computer-aided diagnosis (CAD) model to
Ninety-six consecutive stroke patients were examined by sequential CT scans. Edema in the acute/subacute state and atrophy in the chronic state were evaluated by measuring a frontal horn index, septum-caudate distance and width of cortical sulci. In the acute state the degree of edema increased with
Placental tissue from 160 abortions provoked during the 6th--12th weeks, and 109 spontaneous abortions between weeks 5 and 16, were studied histopathologically on respect of a comparison and quantitation of hydrop degeneration. Significantly more cases of severe hydrop degeneration were observed
Spontaneous rupture of Descemet's membrane occurred in a 76-year-old woman with a 20-year history of Terrien's marginal degeneration. This resulted in corneal hydrops characterized by a clear cystic space filled with aqueous humour that formed between the epithelium and Descemet's membrane. This