dirofilariasis/hypoxia

Dogs with Dirofilaria immitis microfilariae and early radiographic pulmonary artery changes, but without pulmonary hypertension or clinical signs of heartworm disease, were studied. An exaggerated pulmonary hypertensive response was found in these dogs if subjected to 10% inspired oxygen. The mean

Heartworm diseases in dogs is an infectious disease that produces pulmonary hypertension. Dogs with the early vascular changes of heartworm disease, but without the clinical cardiopulmonary signs and pulmonary hypertension, were studied. Dogs with early heartworm were identified that had an

Postadulticide pulmonary hypertension mechanisms and treatment with antihistamines and supplemental oxygen were studied in eight dogs with heartworm disease. To ensure severe postadulticide thromboembolism, additional heartworms (either 20 or 40 into 4 dogs each) were transplanted into naturally

The pulmonary hemodynamic response to unilateral alveolar hypoxia was investigated in pentobarbital-anesthetized dogs with mild heartworm (HW) disease and in dogs free of HW (HWF). Left lung nitrogen ventilation in HWF dogs resulted in a decrease in the fraction of the cardiac output (QT) perfusing

Cardiopulmonary function in seven Beagles (under pentobarbital anesthesia) was determined before they were inoculated with Dirofilaria immitis larvae, at 6 and at 12 months after 50 infective larvae were inoculated, and at 7 and 12 months after adulticide and microfilaricide treatment was begun.

Results of arterial blood gas and acid-base analysis on initial samples prior to therapy were reviewed for 220 dogs admitted to the University of Georgia Veterinary Teaching Hospital. Acidemia or alkalemia was detected in 61 of 220 dogs (28%). The most common acid-base abnormality was metabolic