3 结果
Background Acute coronary syndrome (ACS) is among the leading causes of death(1). Atherosclerosis and coronary thrombotic occlusion are driven by inflammatory pathomechanisms(2). The investigators have shown neutrophilic activation and neutrophil extracellular traps (NETs) at the culprit lesion site
3 Background The use of cardiopulmonary bypass (CPB) during cardiac surgery elicits generalized non-specific systemic inflammatory response syndrome (SIRS) and subsequent activation of the cytokine, complement, and coagulation-fibrinolytic cascades (1). In approximately 11% of the patients SIRS may
Background
Coronary atherosclerosis is responsible for the initiation of acute myocardial infarction with plaque rupture leading to acute coronary thrombosis and myocardial infarction. Current treatment in the acute phase involves re-establishing vessel patency by percutaneous coronary intervention