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orthostatic intolerance/fever

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8 结果

[Change in blood circulation in the orthostatic posture during hyperthermia].

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It has been shown that orthostatic tolerance does not deteriorate if the level of hyperthermia and weight losses are controlled. Orthostatic intolerance develops at a critical level of hyperthermia and water losses. The mechanisms of this effect are described. The results obtained can be used to

Ethanol ingestion prolongs orthostatic intolerance in hyperthermic humans.

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BACKGROUND Both ethanol ingestion and hyperthermia contribute to orthostatic intolerance (OI). OBJECTIVE Since ethanol has been cited as a major risk factor for hyperthermia-related deaths, we hypothesized that ethanol exacerbates OI induced by hyperthermia. METHODS There were seven subjects (four
Many physiological changes associated with spaceflight, including decreases in orthostatic tolerance, exercise capacity, and blood volume have been reported. Orthostatic intolerance is a problem affecting many astronauts immediately postspaceflight. In particular, the relationship between

Pathophysiology of Noncardiac Syncope in Athletes.

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The most frequent cause of syncope in young athletes is noncardiac etiology. The mechanism of noncardiac syncope (NCS) in young athletes is neurally-mediated (reflex). NCS in athletes usually occurs either as orthostasis-induced, due to a gravity-mediated reduced venous return to the heart, or in
Coronavirus disease (COVID-19) is a novel highly contagious infectious disease caused by the coronavirus SARS-CoV2. The virus affects the human respiratory and other systems, and presents mostly as acute respiratory syndrome with fever, fatigue, dry cough, myalgia and dyspnea. The clinical
OBJECTIVE Insulin has opposing influences on blood pressure by simultaneously increasing adrenergic activity and vasodilatating peripheral blood vessels. In this study, we sought to determine whether hyperinsulinemia affects tilt table responses in older adults with type 2 diabetes not complicated
Myalgic encephalomyelitis/chronic fatigue syndrome (ME/CFS) is characterized by unexplained persistent fatigue, commonly accompanied by cognitive dysfunction, sleeping disturbances, orthostatic intolerance, fever, lymphadenopathy, and irritable bowel syndrome (IBS). The extent to which the
The pathogenesis of ME/CFS, a disease characterized by fatigue, cognitive dysfunction, sleep disturbances, orthostatic intolerance, fever, irritable bowel syndrome (IBS), and lymphadenopathy, is poorly understood. We report biomarker discovery and topological analysis of plasma metabolomic, fecal
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