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pneumothorax/hypoxia

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页 1 从 390 结果
Oxygen arterial partial pressure (PaO2) has been measured in 38 patients with idiopathic spontaneous pneumothorax (ISP) and in 20 of them 8-9 days after full expansion of the lung within 1-3 days by aspiration through chest tube drain. The PaO2 was initially 69.0 +/- 9.6 Torr (predicted value 99.6

[Follow-up study of children with hypoxia following pneumothorax].

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A group of 23 children suffering from neonatal hypoxia (pO2 less than 50 mm Hg) due to pneumothorax was examined at an average age of 14 months. Evaluation at this early developmental stage was accomplished by combination of "Motoscopy", "Vojta's postural reactions" and the "Concept of provocation

[Pneumothorax as a cause of acute and recurrent neonatal hypoxia].

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In 191 children suffering from RDS treated by mechanical ventilation or CPAP + PEEP according to the same protocol the incidence of pneumothorax was determined with 23% (44 infants). Mean birthweight and mean gestational age were lower in infants who developed pneumothorax. Pneumothorax occurred

Extracellular fluid volume during pneumothorax and hypoxemia in rabbits.

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This study was designed to test the hypothesis that persistent pneumothorax of greater than or equal to 6 days duration causes a decrease of extracellular fluid volume (ECF). Such changes are of interest as they may be causally related to persistent hypotension that has occurred in humans following
OBJECTIVE To test the hypothesis that, in newborn piglets, the presence of a tension pneumothorax modifies the cardiovascular responses to hypoxia/hypercarbia. METHODS Prospective laboratory study. METHODS Perinatal cardiovascular research laboratory at a university school of medicine. METHODS Seven

Hypoxemia after pneumothorax exsufflation: a case report.

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We describe a 36-year-old patient who was admitted to the emergency ward for acute dyspnea due to a spontaneous pneumothorax. He was successfully drained but shortly after presented a severe hypoxemia due to pulmonary oedema secondary to pulmonary re-expansion. The physiopathology behind this

Intraoperative hypoxia secondary to pneumothorax: The role of lung ultrasound.

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Intraoperative pneumothorax during general anesthesia is a dangerous event. It is a possible cause of sudden intraoperative hypoxia, which can be critical especially in high-risk patients such as those with end-stage heart failure. Early diagnosis and effective treatment are essential. We describe
The effect of dexamethasone (DXM) pretreatment in newborn piglets with experimental pneumothorax (EPT) was studied. Neither low DXM doses nor those administered 1 or 2 h prior to the induction of EPT were found to be effective against its course. In contrast, 5 mg/kg of body wt. of DXM given

THE EFFECT OF ARTIFICIAL PNEUMOTHORAX UPON THE ANOXEMIA OF PNEUMONIA.

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Unilateral pulmonary oedema following re-expansion of pneumothorax.

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Unilateral pulmonary oedema is a rare complication in the routine management of spontaneous pneumothorax. Previous reports have emphasized excessive negative intrapleural pressure, rapid re-expansion of the lungs and bronchial obstruction as major factors in the pathogenesis. We have encountered

Spontaneous pneumothorax in pregnancy and labour.

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A case of spontaneous pneumothorax occurring at the end of labour in a healthy 17-year-old primigravida is described. Its occurrence was accompanied by marked surgical emphysema of the face, neck, arms and thorax. The patient had had previous thyroid surgery and was coincidentally found to have

[Pneumothorax during esophageal endoscopic mucosal resection].

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We report a case of pneumothorax occurring during esophageal endoscopic mucosal resection (EEMR). A 53-year-old man with early esophageal carcinoma was scheduled for EEMR under general anesthesia with artificial ventilation. During the operation, arterial oxygen saturation measured by pulse oximeter

[Electrocardiographic abnormalities in left pneumothorax].

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Pneumothorax is a frequent complication among patients under mechanical ventilation or undergoing various invasive procedures. Its prevalence in intensive care units ranges from 3% to 14%. The electrocardiographic abnormalities described in left pneumothorax include poor precordial R-wave
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