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white/抗真菌药

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Superficial white onychomycosis.

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A study on the incidence and causative organisms of pedal superficial white onychomycosis within several patient populations is presented. Early recognition, debridement, and topical antifungal therapy for several weeks with attention to biomechanical factors should resolve the infection and prevent

[Therapeutic use of white cabbage juice].

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Fifty shades of white: Understanding heterogeneity in white adipose stem cells.

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The excessive expansion of white adipose tissue underlies the global obesity epidemic. However, not all fat is equal, and the impact of heterogeneity on the development and expansion of different adipose depots is becoming increasingly apparent. Two mechanisms are responsible for the growth of

The white dot syndromes.

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OBJECTIVE To review the distinctive and shared features of the white dot syndromes, highlighting the clinical findings, diagnostic test results, proposed etiologies, treatments, and prognosis. METHODS Review. METHODS Review of the literature. RESULTS Common white dot syndromes are reviewed,

White matter injury mechanisms.

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White matter of the brain and spinal cord is susceptible to anoxia, ischemia, trauma and autoimmune attack. Irreversible injury to this tissue can have serious consequences for the overall function of the CNS through disruption of signal transmission. Like neurons, central myelinated axons are

White piedra in children.

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BACKGROUND White piedra is a fungal infection of the hair shaft caused by species of Trichosporon. Rarely has this infection been reported in the United States. Historically, infected individuals required shaving of their hair to achieve clearance of the infection. OBJECTIVE We sought to describe 8

Superficial white onychomycosis revisited.

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BACKGROUND Superficial white onychomycosis (SWO) is a distinct pattern of fungal nail invasion, which is usually treated with topical antifungals. OBJECTIVE This paper presents a case of SWO with deep penetration and records other similar cases. METHODS The clues to deep invasion of the nail plate

Diagnostic and therapeutic strategies of white clot syndrome.

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This study describes our experience with 12 patients with white clot syndrome encountered during a recent 36-month period. The diagnosis was based on the following criteria: (1) development of thrombocytopenia of less than 100,000/mm3 during administration of heparin therapy, (2) normalization of

[Therapeutic management of the Wolff-Parkinson-White syndrome].

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Reentry tachycardia and atrial fibrillation occur in 12 to 36 per cent of patients with Wolff-Parkinson-White (WPW) syndrome. The severity of the arrhythmia depends on the frequency of attacks, on the heart rate in tachycardia and on the condition of the underlying myocardium. Moreover, ventricular

Barriers to the black and white therapeutic relationship.

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[Specialized therapeutic aid in the White Russian SSR].

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["White dot syndromes" in childhood].

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BACKGROUND The term "white dot syndromes" describes a group of heterogeneous inflammatory disorders of the choriocapillaris. They were first described a few decades ago and our knowledge about these variable diseases is very limited, especially in regard to their overall incidence and their

Excitotoxic damage to white matter.

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Glutamate kills neurons by excitotoxicity, which is caused by sustained activation of glutamate receptors. In recent years, it has been shown that glutamate can also be toxic to white matter oligodendrocytes and to myelin by this mechanism. In particular, glutamate receptor-mediated injury to these

White Paper AGA: Functional Dyspepsia.

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Functional dyspepsia remains a major challenge for clinicians with no approved drugs available in the Western World. In October 2016, a meeting was held in Washington, DC, sponsored by the American Gastroenterological Association's Center for Diagnostics and Therapeutics, which included members of

Aging of cerebral white matter.

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White matter (WM) occupies a large volume of the human cerebrum and is mainly composed of myelinated axons and myelin-producing glial cells. The myelinated axons within WM are the structural foundation for efficient neurotransmission between cortical and subcortical areas. Similar to neuron-enriched
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