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anisodamine/hypoxia

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8 結果
The changes of contents of TXB2 and 6-Keto-PGF1a were studied in severely acute hypoxic cultured intra-pulmonary arteriolar smooth muscle cells (PASMCs) under the action of anisodamine. The results demonstrated that the contents of TXB2 and 6-Keto-PGF1a and their ratio were significantly increased
The pulmonary edema (PE) induced by adrenaline (AD) is similar to neurogenic pulmonary edema. Anisodamine (654-2) showed an apparent therapeutic effect on it. The pulmonary wedge arterial pressure (PAWP), carotid arterial pressure (CAP) and blood gases were measured. It was found that in PE group,

[Experimental study on acute hypoxia in newborn rabbits].

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To investigate the hypoxic-ischemic damage of perinatal asphyxia, the acute hypoxic models of newborn rabbits were established and the effects of ligustrazine and anisodamine on pulmonary arterial pressure (PAP), dural blood flow (DBF) and blood gas of the hypoxia rabbits were observed. Acute
OBJECTIVE To study the clinical therapeutic effect of anisodamine on respiratory function after severe brain injury. METHODS Ninety patients with respiratory dysfunction following severe brain injury were divided into two groups: a treatment group (n = 45, treated with routine therapy plus

Anisodamine restores bowel circulation in burn shock.

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In a group of eight burn patients with a mean of 65.3 +/- 17.4 per cent TBSA burn injury (range 50-90 per cent TBSA), accompanied by a mean of 43.5 +/- 18.9 per cent TBSA full-thickness injury, it was shown that the evidence of global hypovolaemia had disappeared at 12 h after the injury following

Chinese herbal medicine increases tissue oxygen tension.

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An herbal medicine widely used in China, anisodamine, was investigated to determine its effect on the tissue PO2 of an important neural tissue, the retina. The anisodamine was injected intravenously over 10 min into anesthetized cats. Although it reduced mean blood pressure an average of 26%,

[Experimental study on combined treatment in smoke inhalation injury].

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Forty-two healthy dogs were randomly divided equally into a control group (CG) and a treated group (TG). All were inflicted with severe smoke inhalation injury and pulmonary was demonstrated. The dogs in TG were treated with injection of shenmai zhusheye, ketoprofen, anisodamine, sodium aesculin,
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