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oligohydramnios/nicotine

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12 結果
OBJECTIVE Morphological lesions in placenta and changed metal distribution through placenta, which were observed in pregnancy exposed to tobacco smoke, could cause alternations in lysosomal enzymes secretion to amniotic fluid. METHODS Assessment of total activity and molecular forms of
The purpose of the study was to estimate the concentration of the following proteins: metallothionein, beta-2-microglobuline (BMG), interleukin 6 (II-6) in the amniotic fluid of smoking women during their pregnancy complicated by idiopathic oligohydramnios or premature rupture if the membranes
To assess the exposure to tobacco smoke in pregnant women with oligohydramnios, idiopathic or caused by premature rupture of the membranes (PROM), cotinine concentrations were measured, using enzyme-like immunosorbent assay (ELISA). In women with idiopathic oligohydramnios (22-31 weeks of
OBJECTIVE The ingredients of tobacco smoke have the impact on uterine blood vessels. They caused vascular insufficiency of placenta during development of gestation and changes in placental tissue and fetal membranes. It manifest changeable metal permeability and others symptoms. METHODS Assessment
The aim of the present study was to assess the activity of membrane enzymes: alanine aminopeptidase (AAP), gamma-glutamyltransferase (GGT) and trehalase in amniotic fluid of women who smoke cigarettes during pregnancy complicated by idiopathic oligohydramnios or premature rupture of the membranes
OBJECTIVE The aim of the study was analysis of neonatal status from pregnancies complicated oligohydramnios and from pregnancies complicated premature rupture of the membranes (PROM). METHODS Authors analyzed 15 newborns from pregnancies with oligohydramnios diagnosed and 15- from pregnancies

[Oligohydramnios. Diagnosis. Etiology. Prognosis].

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We have studied 68 cases of severe oligohydramnios and compared them with the published literature on the aetiology, pathology, diagnosis, complications and prognosis. The principal aetiological features have been malformations of the fetal urinary tracts, intra-uterine growth retardation, high

Fetal and early postnatal life roots of asthma.

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The origins of asthma might be traced back to events occurring during fetal life. Reduced lung development has been shown to be a risk factor both for viral induced wheeze and allergic asthma. The evidence for a causal relationship between exposure to environmental tobacco smoke, chemical domestic

[Risk factors associated with preterm birth in a second level hospital].

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BACKGROUND Preterm birth is one of the biggest problems in obstetrics and gynecology, given that it has an incidence of 10-11%. The objective was to identify the risk factors associated with a preterm birth. METHODS A retrospective, observational, transversal and analytic case-control study was

Epidemiological Risk Factors and Perinatal Outcomes of Congenital Anomalies.

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Objectives To identify the epidemiological risk factors for congenital anomalies (CAs) and the impact of these fetal malformations on the perinatal outcomes. Methods This prospective cohort study comprised 275 women whose fetuses had CAs. Maternal variables to establish potential risk factors for
BACKGROUND The U.S. Natality database from 2001 and 2002 was used to investigate the relationship between maternal cigarette smoking during pregnancy and the risk of having a child with polydactyly, syndactyly, or adactyly. METHODS The records of 6,839,854 live births were examined to identify 5171

[Reverse flow in fetal vessels and perinatal events].

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BACKGROUND/PATIENTS: A reverse flow in the umbilical artery and/or fetal aorta is associated with a higher perinatal and neonatal mortality. 30 fetuses showed a reverse flow using pulsed wave Doppler sonography (group I). A matched-pair control group including 30 fetuses with the same gestational
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